Downregulation of androgen receptors in response to supraphysiological levels of androgen is a myth with not only no proof for it, but no evidence except in a few cases where testing methods are shown to be flawed.
There isn’t an issue of receptors being clean or not.
It can be the case (not that it always will be the case) that Dianabol Methandienone can be used while maintaining normal LH. I only know of that being found where it has been used morning-only rather than in divided doses.
I used to advocate modest morning-only use in the early recovery phase but stopped doing so on recoveries fairly clearly averaging slower when this was done.
But where steroids weren’t used previously, even 50 mg/day morning only can still allow normal LH production. I’m not saying it always will but it can. That is even without an antiaromatase.
One problem with bridging use is, If one is using orals off cycle, then how does the liver get a break from the alkylateds if planning to use them during cycles as well? Is the off-cycle use going to be at the cost of sacrificing their use during cycles?
But if wanting to try this use, I would test estrogen level, keep it low normal, and see what happens with T levels.
In this instance I think it is not necessary to measure LH because at least in my previous experience with this (haven’t done any such testing in a long time) Dianabol did not seem to be picked up by the tests as falsely being testosterone.